The risk of acute pancreatitis in patients using cardiovascular drugs has been extensively studied in the European study on drug-induced acute pancreatitis pomegranate juice blood pressure medication buy triamterene 75mg with visa. The risk increased with higher daily doses and was highest in the first six months of therapy (Eland et al blood pressure pills generic triamterene 75 mg without a prescription. Histamine H2 receptor antagonists cimetidine and ranitidine have been reported to pulse pressure 66 quality triamterene 75 mg cause drug-induced pancreatitis in several case reports without an evidence of rechallenge or a consistent latency pulse pressure change with exercise purchase triamterene with visa. Some experimental findings also indicate the possible causative relationship, whilst others deny it. On the other hand, a previous, much larger and better designed study brought no evidence for this suspicion (Eland et al. This phenomenon was probably even more pronounced in a newer group of agents with similar effects, dipeptidyl peptidase-4 inhibitors. A considerable effort has been made to refute this connection, which is, of course, in the interest of the manufacturers. Here is yet another example of a negative result in a pharmacoepidemiological study. Again, the probable reason lies in an extremely small proportion of drug-induced cases in total numbers of acute pancreatitis, which of course cannot influence the overall risk in high-risk populations. Available clinical case reports or series are usually too outdated to rely on the information contained (Bartholomew, 1970), but experimental studies on the effects of scorpion toxin are very interesting. Concurrent stimulation of pancreatic secretion and contraction of the sphincter of Oddi have been demonstrated in the late 1970s. Rare reports on pancreatitis caused by adder bite (venom containing neurotoxic phospholipase A2) or even blue-ringed octopus bite (venom containing tetrodotoxin) have been published. Aside from alcohol, another addictive substance often mentioned in association with acute pancreatitis is marijuana, abused by smoking. A smaller series of marijuana-induced pancreatitis cases was reported by Wargo et al. Interestingly, stimulation of cannabinoid receptors was found to be a protective mechanism during experimental pancreatitis. This is yet another example of ambivalent behavior of some xenobiotics towards the pancreatic tissue. Diagnostics, disease course and management Among the reasons why the real incidence of drug-induced acute pancreatitis is still not known, the difficulties in diagnosis are probably most important. Milder cases of pancreatic injury are often missed because serum amylase and lipase estimations are not part of the metabolic profile obtained during a routine health checkup and abdominal pain is often attributed to underlying diseases. The first criterion seems to be easy to achieve until we remember that monotherapy in our patients becomes more and more scarce. Use of the classification systems mentioned above may be very useful for that purpose. Excluding all other causes of the disease is also not so straightforward in many cases of acute pancreatitis. The validity of diagnosis may depend on the equipment available and even more on the experience of the medical staff. Discontinuation of oral therapy is a natural part of any management of acute pancreatitis. In patients treated by multiple pharmacotherapy, it is impossible to decide which medication withdrawal led to a resolution of the symptoms and laboratory findings. In these cases, acute pancreatitis is usually diagnosed within several days from drug administration. Due to the character of the disease and ethical considerations, deliberate, repeated administration of suspect drug to induce a new episode of acute pancreatitis is not possible. An exception is the use of essential drugs in cases where the benefits outweigh the risks. A simplified algorithm for diagnosing drug-induced pancreatitis is given in Figure 1. The suspected drug etiology should be considered after the exclusion of more common causes of illness. A detailed medication history documentation is obvious as well as the determination of suspicious substances. There is no evidence for preferring one of these systems, so it is possible to use both, mainly if there is a difference between them in classifying a specific suspicious agent. Using these classification systems may improve the quality of information for further patient treatment and further processing of the event for scientific or pharmacovigilance purposes. Level of Characteristics probability Certain A clinical event, including a laboratory test abnormality, that occurs in a plausible time relation to drug administration, and which cannot be explained by concurrent disease or other drugs or chemicals the response to withdrawal of the drug (dechallenge) should be clinically plausible the event must be definitive pharmacologically or phenomenologically using a satisfactory rechallenge procedure if necessary Probable A clinical event, including a laboratory test abnormality, with a reasonable time relation to administration of the drug, unlikely to be attributed to concurrent disease or other drugs or chemicals, and which follows a clinically reasonable response on withdrawal (dechallenge) Rechallenge information is not required to fulfill this definition Possible A clinical event, including a laboratory test abnormality, with a reasonable time relation to administration of the drug, but which could also be explained by concurrent disease or other drugs or chemicals Information on drug withdrawal may be lacking or unclear Unlikely A clinical event, including a laboratory test abnormality, with a temporal relation to administration of the drug, which makes a causal relation improbable, and in which other drugs, chemicals, or underlying disease provide plausible explanations Conditional / A clinical event, including a laboratory test abnormality, reported as an unclassified adverse reaction, about which more data are essential for a proper assessment or the additional data are being examined Unassessable / A report suggesting an adverse reaction that cannot be judged, because unclassifiable information is insufficient or contradictory and cannot be supplemented or verified Table 3. Of course, severe cases tend to be more often 30 Acute Pancreatitis reported both in the literature and in spontaneous pharmacovigilance reports. In the disease management, there are no specific issues concerning drug-induced pancreatitis, with an exception of an immediate withdrawal of the suspected drug. A difficult question is how to reintroduce medication if the causative agent is not unambiguously identified. We recommend not introducing all withdrawn drugs at the same time to distinguish the cause of a possible flare-up. The most suspected drugs should be substituted by their analogs with a different chemical structure. Secondary prevention consists of avoiding the drug which caused the episode of acute pancreatitis. Rechallenge of such an agent is justified only if its benefits outweigh the risks, as discussed above. Future research Given how inadequate the current state of knowledge on drug-induced pancreatic injury is, the area for further research in this field is remarkably wide. The majority of the knowledge on the topic has been obtained from case reports or their series. These will remain a major source of information, so it is necessary to improve their informative value substantially. Provide the age and sex of the patient, along with the indication for treatment with a drug; provide the dose and frequency of medication; b. Document a definite case of pancreatitis based on current diagnostic guidelines; c. Provide information on the time course between initiation of drug and onset of pancreatitis; d. Exclude the most common causes of pancreatitis; document a positive response to withdrawal of medication;. Higher level of knowledge may be obtained by performing multicenter studies targeted at the etiology of non-alcoholic, non-biliary pancreatitis. Several thousands of acute pancreatitis cases must be involved in these studies to reveal the actual occurrence of drug induced pancreatitis. Any new pharmacoepidemiological study on this topic would be useful, but to improve the validity of its outcomes, substantially better input data are required. For this purpose, it would be optimal that each single case of acute pancreatitis included in such a study be documented according to the above principles. An obvious field for this research is the issue of diseases with a high Acute Pancreatitis Induced by Drugs 31 incidence of this disorder. Another issue is the experimental pharmacological research of mechanisms by which xenobiotics can damage the pancreatic tissue as well as the common mechanisms of immune-mediated tissue injury caused by drugs. Any substantial progress in this research can contribute to a progress in two scientific challenges: recognizing the nature of more frequent causes of acute pancreatitis and also recognizing the cause and pathogenesis of idiosyncratic adverse drug reaction. Epidemiological studies show a very wide range of its incidence, but at least the absolute number of its cases is undoubtedly increasing. We are able to identify the drugs with the greatest risk and populations at risk, but the absolute risk for medication users is still very low. A better understanding of drug mediated pancreatic injury can also help to understand the etiology of more common types of acute pancreatitis. Research in drug-induced acute pancreatitis is both a challenge and an opportunity to improve the collaboration of gastroenterology and clinical pharmacology. Introduction Evidence accumulated for the past two decades leads to the conclusion that obesity enhances the development of acute pancreatitis and worsens its clinical course. We will try to give an answer to this issue by presenting the scientific data accumulated thus far. According to the definition, one should calculate the total amount of body fat a person has and deduct the normal amount of fat from it.
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The lesions occur chiefly on the extensor surfaces of the joints and other areas exposed to blood pressure monitor chart printable purchase triamterene 75 mg amex trauma heart attack high blood pressure buy discount triamterene 75 mg on-line. The severe dystrophic type that leads to pulse pressure wave qrs complex triamterene 75 mg cheap scarring may also produce conjunctival scars similar to arteria ductus deferentis buy 75 mg triamterene with visa those seen in dermatitis herpetiformis and mucous membrane pemphigoid. The signs are papillary hypertrophy of the upper tarsus, redness of the superior bulbar conjunctiva, thickening and keratinization of the superior limbus, epithelial keratitis, recurrent superior filaments, and superior micropannus. In about 50% of cases, the condition has been associated with abnormal function of the thyroid gland. In severe cases, one may consider 5-mm resection of the perilimbal superior conjunctiva. Recent studies have shown an underlying type 1 plasminogen deficiency in patients suffering from ligneous conjunctivitis. Iritis can also occur, but tends to be 245 a late complication. There is no satisfactory treatment, although nonsteroidal anti-inflammatory agents may be effective. As well as bilateral nonpurulent conjunctivitis, the clinical manifestations include fever, oropharyngeal abnormalities, erythema of the palms or soles, edema of the hands or feet, rash on the trunk, and cervical lymphadenopathy. As the disease progresses, the chemosis increases, and in advanced cases, the chemotic conjunctiva may extrude between the lids (Figure 523), leading to corneal exposure that is exacerbated by any proptosis. Graves ophthalmopathy with marked chemosis leading to conjunctival prolapse, keratinization, and injection and inadequate corneal wetting. On examination, a mild conjunctivitis is found that is less severe than suggested by the symptoms. Gout may also be associated with episcleritis or scleritis, iridocyclitis, keratitis, vitreous opacities, and retinopathy. Treatment is aimed at controlling the gouty attack with colchicine and allopurinol. The nature and source of the conjunctivitis in both instances are often missed until the lacrimal system is investigated. The source of the condition is often missed unless the characteristic hyperemic, pouting punctum is noted. Expression of the canaliculus (upper or lower, whichever is involved) is curative provided the entire concretion is removed. Candida grows readily on ordinary culture media, but almost all of the infections are caused by A israelii, which requires an anaerobic medium. They appear as yellow nodules on both sides of the cornea (more commonly on the nasal side) in the area of the palpebral aperture (Figure 524). The nodules, consisting of hyaline and yellow elastic tissue, rarely increase in size, but inflammation is common. In general, no treatment is required, but in certain cases of pingueculitis, weak topical steroids (eg, prednisolone 0. It is thought to be an irritative phenomenon due to ultraviolet light, drying, and windy environments, since it is common in persons who spend much of their lives out of doors in sunny, dusty, or sandy, windblown surroundings. In the cornea, there is replacement of Bowmans layer by hyaline and elastic tissue. If the pterygium is enlarging and encroaches on the pupillary area, it should be removed surgically along with a small portion of superficial clear cornea beyond the area of encroachment. Conjunctival autograft at the time of surgical excision has been shown to reduce the risk of recurrent disease. They are merely dilated lymph vessels, and no treatment is indicated unless they are irritating or cosmetically objectionable. Usually observed as an isolated entity at birth, the condition is thought to be due to a congenital defect in the lymphatic drainage of the conjunctiva. It has been observed in chronic hereditary lymphedema of the lower extremities (Milroys disease) and is thought to be an ocular manifestation of this disease rather than an associated anomaly. Its sudden onset and bright-red appearance usually alarm the patient (Figure 527). The hemorrhage is caused by rupture of a small conjunctival vessel, sometimes preceded by a bout of severe coughing or sneezing. In rare instances, if the hemorrhages are bilateral or recurrent, the possibility of blood dyscrasias should then be ruled out. In its narrow and commonly used sense, however, it refers to a conjunctival infection, chiefly gonococcal, that follows contamination of the babys eyes during its passage through the mothers cervix and vagina or during the postpartum period. Because gonococcal conjunctivitis can rapidly cause blindness, the cause of all cases of ophthalmia neonatorum should be verified by examination of smears of exudate, epithelial scrapings, cultures, and rapid tests for gonococci. Gonococcal neonatal conjunctivitis causes corneal ulceration and blindness if not treated immediately. Chlamydial neonatal conjunctivitis (inclusion blennorrhea) is less destructive but can last months if untreated and may be followed by pneumonia. The time of onset is important but not entirely reliable in clinical diagnosis since the two principal types, gonorrheal ophthalmia and inclusion blennorrhea, have widely differing incubation periods: gonococcal disease, 23 days; and chlamydial disease, 512 days. Treatment for neonatal gonococcal conjunctivitis is with ceftriaxone, 125 mg as a single intramuscular dose; a second choice is kanamycin, 75 mg intramuscularly. To treat chlamydial conjunctivitis in newborns, erythromycin oral suspension is effective at a dosage of 50 mg/kg/d in four divided doses for 2 weeks. Herpes simplex keratoconjunctivitis is treated with acyclovir, 30 mg/kg/d in three divided doses for 14 days. Other types of neonatal conjunctivitis are treated with erythromycin, gentamicin, or tobramycin ophthalmic ointment four times daily. Crede 1% silver nitrate prophylaxis is effective for the prevention of gonorrheal ophthalmia but not inclusion blennorrhea or herpetic infection. The slight chemical conjunctivitis induced by silver nitrate is minor and of short duration. Accidents with concentrated solutions can be avoided by using wax ampules specially prepared for Crede prophylaxis. The most common cause is cat-scratch disease, but there are many other causes, including Mycobacterium tuberculosis, Treponema pallidum, Francisella tularensis, Pasteurella (Yersinia) pseudo-tuberculosis, C trachomatis serovars L1, L2, and L3, and C immitis. Conjunctival Cat-Scratch Disease this protracted but benign granulomatous conjunctivitis is found most commonly in children who have been in intimate contact with cats. The child often runs a low-grade fever and develops a reasonably enlarged preauricular node and one or more conjunctival granulomas. The disease appears to be caused by a slender pleomorphic gram-negative bacillus (Bartonella [formerly Rochalimaea] henselae), which grows in the walls of blood vessels. With special stains, this organism can be seen in biopsies of conjunctival tissue. The organism closely resembles Leptotrichia buccalis, and the disease was previously known as leptotrichosis conjunctivae (Parinaud conjunctivitis). The organism is commonly found in the mouth in humans and always in the mouth in cats. The eye may be contaminated by saliva on the childs fingers or by cat saliva on the childs pillow. The disease is self-limited (without corneal or other complications) and resolves in 23 months. The conjunctival nodule can be excised; in the case of a solitary granuloma, this may be curative. Systemic tetracyclines may shorten the course but should not be given to children under 7 years of age. Conjunctivitis Secondary to Neoplasms (Masquerade Syndrome) When examined superficially, a neoplasm of the conjunctiva or lid margin is often misdiagnosed as a chronic infectious conjunctivitis or keratoconjunctivitis. Since the underlying lesion is often not recognized, the condition has been referred to as masquerade syndrome. The masquerading neoplasms on record are conjunctival capillary carcinoma, conjunctival carcinoma in situ, infectious papilloma of the conjunctiva, sebaceous gland carcinoma, and verrucae. Verrucae and molluscum tumors of the lid margin may desquamate toxic tumor material that produces a chronic conjunctivitis, keratoconjunctivitis, or (rarely) keratitis alone. Readers are referred to other sections of this chapter for information about inflammatory and 254 degenerative lesions of the conjunctiva (eg, pingueculum and pterygium) that can simulate conjunctival neoplasms. They may enlarge slowly but have little or no invasive potential and no metastatic capability. Hamartomas are congenital tumors composed of normal or near normal cells and tissues that occur normally at that anatomic site but are present in abnormally excessive amounts.
It is necessary to blood pressure 4080 generic triamterene 75mg without a prescription assume birth trauma essential hypertension triamterene 75 mg on line, anoxia or hypoxia as a condition intervening between mental retardation and the underlying cause prehypertension 126 cheap triamterene 75 mg, premature separation of placenta blood pressure band triamterene 75 mg low cost. As a guide to the acceptability of sequences in the application of the General Principle and the selection rules, the following relationships should be regarded as highly improbable: a. The preceding list does not cover all highly improbable sequences, but in other cases the General Principle should be followed unless otherwise indicated. Acute or terminal circulatory diseases reported as due to malignant neoplasm, diabetes or asthma should be accepted as possible sequences in Part I of the certificate. The following conditions are regarded as acute or terminal circulatory diseases: I21-I22 Acute myocardial infarction I24. A diagnostic term that contains one of the following adjectival modifiers indicates the condition modified has undergone certain changes and is considered to be a one-term entity. Code for Record I (a) Hemorrhagic cardiomyopathy I428 Code to the category for other cardiomyopathies (I428). The Classification does provide a code, I428, for Other cardiomyopathies in Volume 1. Code bronchiectasis only, since there is no provision in the Classification for coding other bronchiectasis. Alzheimer dementia: Consider the following terms as one term entities and code as indicated: When reported as: Code Endstage Alzheimer, senile dementia Senile dementia, Alzheimer G301 Senile dementia, Alzheimer type Senile dementia of the Alzheimer When reported as: Code Alzheimer, dementia Alzheimer; dementia Alzheimer disease (dementia) Dementia Alzheimer Dementia, Alzheimer Dementia Alzheimer Dementia, Alzheimer type Dementia of Alzheimer G309 Dementia Alzheimer type Dementia; Alzheimer type Dementia, probable Alzheimer (disease) Dementia syndrome, Alzheimer type Endstage dementia (Alzheimer) 2. Multiple one-term entity: A multiple one-term entity is a diagnostic entity consisting of two or more contiguous words on a line for which the Classification does not provide a single code for the entire entity but does provide a single code for each of the components of the diagnostic entity. Consider as a multiple one-term entity if each of the components can be considered as separate one-term entities, i. Codes for Record I (a) Hypertensive arteriosclerosis I10 I709 Code to hypertension (I10). Code for Record I (a) Hypertensive myocardial ischemia I259 Code to myocardial ischemia (I259). Adjective reported at the end of a diagnostic entity Code an adjective reported at the end of a diagnostic entity as if it preceded the entity. Codes for Record I (a) Arteriosclerosis, hypertensive I10 I709 Code to hypertension (I10). If an adjectival modifier is reported with more than one condition, modify only the first condition. Codes for Record I (a) Arteriosclerotic nephritis and cardiomyopathy I129 I429 Code to arteriosclerotic nephritis (I129). If an adjectival modifier is reported with one condition and more than one site is reported, modify all sites. Codes for Record I (a) Arteriosclerotic cardiovascular and cerebrovascular disease I250 I672 Code to arteriosclerotic cardiovascular disease (I250). The modifier is applied to both conditions, but in this case the selected cause is not modified by the other condition on the record. When an adjectival modifier precedes two different diseases that are reported with a connecting term, modify only the first disease. Codes for Record I (a) Arteriosclerotic cardiovascular disease and cerebrovascular disease I250 I679 Code to arteriosclerotic cardiovascular disease (I250). When one medical entity is reported followed by another complete medical entity enclosed in parenthesis, disregard the parenthesis and code as separate terms. Consider line (b) as two separate terms, both of which are complete medical entities. When the adjectival form of words or qualifiers are reported in parenthesis, use these adjectives to modify the term preceding it. Codes for Record I (a) Collapse of heart I509 (b) Heart disease (rheumatic) I099 Code to rheumatic heart disease (I099). If the term in parenthesis is not a complete term and is not a modifier, consider as part of the preceding term. Code for Record I (a) Metastatic carcinoma (ovarian) C56 Code to primary ovarian carcinoma (C56). Plural form of disease Do not use the plural form of a disease or the plural form of a site to indicate multiple. Codes for Record I (a) Cardiac arrest I469 (b) Congenital defects Q899 Code to congenital defect (Q899); do not code as multiple (Q897). Implied disease When an adjective or noun form of a site is entered as a separate diagnosis, i. Codes for Record I (a) Coronary I251 (b) Hypertension I10 (c) Code to coronary disease (I251). Line I(a) is coded as coronary disease since coronary hypertension is not indexed. Consider the site, renal, to be a part of the condition that immediately follows it on line b, since Hypertension, renal is indexed. Non-traumatic conditions Consider conditions that are usually but not always traumatic in origin to be qualified as non-traumatic when reported due to or on the same line with a disease. I (a) Fat embolism I749 (b) Pathological fracture M844 Code line I(a) as non-traumatic since reported due to a disease. Generally, it may be assumed that such a condition was of the same site as another condition if the Classification provides for coding the condition of unspecified site to the site of the other condition. These coding principles apply whether or not there are other conditions reported on other lines in Part I. Conditions of unspecified site reported on the same line (1) When conditions are reported on the same line with or without a connecting term that implies a due to relationship, assume the condition of unspecified site was of the same site as the condition of a specified site. Codes for Record I (a) Aspiration pneumonia J690 (b) Cerebrovascular accident due to I64 (c) thrombosis I633 Code to cerebral thrombosis (I633). Since thrombosis (of unspecified site) is reported on the same line with a condition of a specified site, relate to the specified site. Since infarction (of unspecified site) is reported on same line with two conditions of specified sites, relate to the specified site immediately preceding the condition. Conditions of unspecified site reported on a separate line (1) If there is only one condition of a specified site reported on the line above or below it, code to this site. Codes for Record I (a) Cholecystitis K819 (b) Calculus K802 Code to calculus of gallbladder with other cholecystitis (K801). Codes for Record I (a) Intestinal fistula K632 (b) Obstruction K566 (c) Adhesions of peritoneum K660 Code to intestinal adhesions with obstruction (K565). Since the Classification does not provide a code for obstruction of the peritoneum, relate to the site reported on the line above (intestinal). Since the thrombosis is classified to both sites (reported above and below), do not relate. It is acceptable to relate conditions not reported as the first condition on a line to the line below. Codes for Record I (a) Gastrointestinal hemorrhage K922 (b) Peptic ulcer K279 Code to peptic ulcer with hemorrhage (K274). Codes for Record I (a) Peritonitis K659 (b) Ulcer K279 Code to peptic ulcer (K279). When hernia (K40-K46) is reported with disease(s) of unspecified site(s), relate the disease of unspecified site to the intestine. Codes for Record I (a) Hernia with obstruction K469 K566 Code to hernia with obstruction (K460). Codes for Record I (a) Calculus with pyelonephritis N209 N12 Code to urinary calculus (N209). When arthritis (any type) is reported with Contracture code contracture of the site Deformity code deformity acquired of the site If no site is reported or if site is not indexed, code contracture or deformity, joint. Codes for Record I (a) Phlebitis I809 (b) Deformities M219 (c) Osteoarthritis lower limbs M199 Code to osteoarthritis lower limbs (M199). Relate a condition of unspecified site to the complete term of a multiple site entity. If it is not indexed together, relate the condition to the site of the complete indexed term. Codes for Record I (a) Cardiorespiratory arrest with I469 I509 (b) insufficiency Code to heart failure (I509). Since cardiorespiratory arrest is indexed to a heart condition, relate insufficiency to heart. Codes for Record I (a) Renal failure N19 (b) Vasculitis I778 Code Vasculitis, kidney (I778). Do not relate conditions classified to R00-R99 except: Gangrene and necrosis R02 Hemorrhage R5800 Stricture and stenosis R688 Codes for Record I (a) Pneumonia with gangrene J189 J850 Code to gangrene of lung (J850).
A treatise on the effects and various preparations of lead : particularly of the extract of saturn heart attack and water order 75 mg triamterene visa. 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W E 550 M 6845a 1998 An abridgem ent of the practice of m idw ifery : and a set of anatom ical tables w ith explanations Sm ellie, W illiam, W Z 270 S638s 1786 An account of som e of the m ost im portant diseases peculiar to w om en G ooch, Robert, W Q G 645a 1848 An account of the bilious rem itting yellow fever, as it appeared in the city of Philadelphia, in the year 1793 Rush, Benjam in, W Z 260 R952a 1794 An account of the bilious rem itting yellow fever, as it appeared in the city of Philadelphia, in the year 1793 Rush, Benjam in, W Z 260 R952a 1794 An account of the institution and progress of the College of physicians of Philadelphia during a hundred years, from January, 1787. An account of the m alignant fever, lalely [sic] prevalent in the city of New York H ardie, Jam es, W Z 270 H 262a 1799 *These volum es are not available for circulation. An Am erican textbook of gynecology, m edical and surgical, for practitioners and students. W O K26a 1893 An analysis of physiology : being a condensed view of its m ost im portant facts and doctrines, designed especially for the use of Reese, John J. Q T R329a 1847 students An analysis of physiology : being a condensed view of its m ost im portant facts and doctrines, designed especially for the use of Reese, John J. Q T R329a 1852 students An analytical com pendium of the various branches of m edical science : for the use and exam ination of students Neill, John, W B N412a 1848 An analytical com pendium of the various branches of m edical science, for the use and exam ination of students, Neill, John, W B N412a 1852 An atlas of hum an anatom y : illustrating m ost of the ordinary dissections and m any not usually practised by the student G odlee, Rickm an John, Q S G 586a 1880 An atlas of the central nervous system and cranial nerves, H irschfeld, Ludovic, W L H 669a 1890 An easy and exact m ethod of curing the venereal disease. W Z 260 P9665e 1748 by w ay of dialogue betw een physician and patient, for the use and instruction of all unfortunate persons w ho m ay labour under that disorder. An elem entary system of physiology, com prising a com plete view of the present state of the science, including an account of all Bostock, John, Q T B747e 1836 the m ost im portant facts and observations, and analyses of the principal theories and hypotheses. An elem entary treatise on hum an anatom y Leidy, Joseph, Q S L527e 1861 An elem entary treatise on hum an physiology : on the basis of the Precis elem entaire de physiologie M agendie, Francois, Q T M 192e 1845 An elem entary treatise on m idw ifery: or Principles of tokology and em bryology. W Q V444t 1831 An elem entary treatise on m idw ifery; or, Principles of tokology and em bryology. W E D4815e 1840 An essay on curvatures and diseases of the spine : including all the form s of spinal distortion : to w hich the Fothergillian gold Bam pfield, R. W E B211e 1845 m edal w as aw arded by the M edical Society of London An essay on diseases incidental to Europeans in hot clim ates. To Lind, Jam es, W Z 260 L742es 1792 w hich is added, an appendix concerning interm ittent fevers. And a sim ple and easy w ay to render sea w ater fresh, and to prevent a scarcity of provisions in long voyages at sea. H uxham, John, W Z 260 H 987e 1779 An essay on hydrocephalus acutus, or dropsy in the brain Cheyne, John, W Z 270 C531eh 1814 An essay on the anim al oeconom y : together w ith observations upon the sm all pox H elvetius, Jean Claude Adrien, W Z 260 H 371i 1723 An essay on the causes of the variety of com plexion and figure in the hum an species. To w hich are added, anim adversions on Sm ith, Sam uel Stanhope, W Z 270 S659e 1810 certain rem arks m ade on the first edition of this essay, by M r. Charles W hite, in a series of discourses delivered before the Literary and Philosophical Society of M anchester in England. An essay on the disease called yellow fever : w ith observations concerning febrile contagion, typhus fever, dysentery, and the Bancroft, Edw ard Nathaniel, W Z 270 B213e 1820 plague, partly delivered as the G ulstonian lectures, before the College of Physicians, in the years 1806 and 1807 An essay on the m alignant pestilential fever introduced into the W est Indian Islands from Boullam, on the coast of G uinea, as it Chisholm, Colin, W Z 270 C542e 1799 appeared in 1793 and 1794 An essay on the m eans of lessening pain, and facilitating certain cases of difficult parturition Dew ees, W illiam P. Reid, Thom as, W Z 260 R358e 1798 An essay on the nature of scrofula, w ith evidence of its origin from disorder of the digestive organs. W alker is chairm an : prepared for the inform ation of said Com m ittee An experim ental inquiry into the law s of the vital functions : w ith som e observations on the nature and treatm ent of internal Philip, Alexander Philip W ilson, W Z 270 P552e 1818 diseases An experim ental inquiry into the nature, cause, and varieties of the arterial pulse, and into certain other properties of the larger Parry, Caleb H illier, W G P264e 1816 arteries, in anim als w ith w arm blood. An experim ental investigation into the functions of the eighth pair of nerves : or the glossopharyngeal, pneum ogastric, and Reid, John, W L R356e 1840 spinal accessory An exposition of quackery and im posture in m edicine; being a popular treatise on m edical philosophy. Ticknor, Caleb, W Z 310 T557p 1839 An exposition of the signs and sym ptom s of pregnancy, the period of hum an gestation, and the signs of delivery. W Q 202 M 788e 1839 An historical account of the clim ates and diseases of the United States of Am erica : and of the rem edies and m ethods of Currie, W illiam, W Z 270 C976h 1792 treatm ent, w hich have been found m ost useful and efficacious, particularly in those diseases w hich depend upon clim ate and situation : collected pricipally from personal observation, and the com m unications of physicians of talents and experience, residing in the several states An illustrated encyclop dic m edical dictionary : being a dictionary of the technical term s used by w riters on m edicine and the Foster, Frank P. W 13 F754i 1890 collateral sciences, in the Latin, English, French and G erm an languages An illustrated encyclop dic m edical dictionary : being a dictionary of the technical term s used by w riters on m edicine and the Foster, Frank P. Tanner, Thom as H aw kes, W B T167i 1882 An inquiry into the degree of certainty in m edicine : and into the nature and extent of its pow er over disease Bartlett, Elisha, W B289i 1848 An inquiry into the nature and origin of m ental derangem ent : com prehending a concise system of the physiology and pathology Crichton, Alexander, W Z 260 C925i 1798 of the hum an m ind. An inquiry into the nature and treatm ent of diabetes, calculus, and other affections of the urinary organs; w ith rem arks on the Prout, W illiam, W J 100 P968o 1826 im portance of attending to the state of the urine in organic diseases of the kidney and bladder: and som e practical rules for determ ining the nature of the disease from the sensible and chem ical properties of that secretion. Six lectures on the m ethod of exam ining patients; percussion, auscultation, the use of the Bennett, John H ughes, W B B471i 1853 m icroscope, and the diagnosis of skin diseases. H enry Q Z 4 G 798i 1876 An introduction to the study of hum an anatom y Paxton, Jam es, Q S P342I 1837 *These volum es are not available for circulation. Q Z M 848o 1838 Anaesthesia, or the em ploym ent of chloroform and ether in surgery, m idw ifery, etc. A com plete series of anatom ical questions w ith answ ers; the answ ers arranged so as to form an Q S 518 A535a 1811 elem entary system of anatom y, and intended as preparatory to exam inations at Surgeons H all; to w hich are annexed tables of the bones, m uscles, and arteries. Anatom ical investigations, com prising descriptions of various fasciae of the hum an body : the discoveries of the m anner in w hich G odm an, John D. Q S G 587an 1824 the pericardium is form ed from the superficial fascia, the capsular ligam ent of the shoulder joint from the brachial fascia, and the capsular ligam ent of the hip joint from the fascia lata : to w hich is added an account of som e irregularities of structure and m orbid anatom y : w ith a description of a new anatom ical table Anatom ical Rem em brancer. Benedikt, M oriz, H V B463a 1881 Anatom ie und Physiologie des Nervensystem s des M enschen und der W irbelthiere, m it pathologischen Beobachtungen und m it Longet, F. Anatom ie und Physiologie des Nervensystem s des M enschen und der W irbelthiere, m it pathologischen Beobachtungen und m it Longet, F. Anatom y : descriptive and surgical G ray, H enry, Q S G 779a 1862 Anatom y of the hum an body* G ray, H enry, Q S 4 G 779an 1924 Anatom y, descriptive and surgical G ray, H enry, Q S G 779a 1867 Anatom y, descriptive and surgical G ray, H enry, Q S G 779a 1870 Anatom y, descriptive and surgical G ray, H enry, Q S G 779a 1878 Anatom y, descriptive and surgical G ray, H enry, Q S G 779a 1887 Anatom y, descriptive and surgical. Ediderunt Academ ia M edicinae NovaEboracensis et Biobliotheca Universitatis Vesalius, Andreas, W Z 290 V575ic 1935 M onacensis. Anim al and vegetable physiology, considered w ith reference to natural theology Roget, Peter M ark, Q H 306 R732 1839 Anim al and vegetable physiology, considered w ith reference to natural theology Roget, Peter M ark, Q H 306 R732 1839 Anim al chem istry : w ith reference to the physiology and pathology of m an Sim on, Johann Franz, Q U S595a 1846 Anim al physiology Carpenter, W illiam Benjam in, Q T C297a 1851 Annales m edico psychologiques. W A P415 Annual report of the trustees of the city hospital of the city of W orcester for the year ending. W orcester City H ospital W X W 921a Annual report of the trustees of the city hospital of the city of W orcester for the year ending. W orcester City H ospital W X W 921a Annual reports on diseases of the chest, Dobell, H orace, W F D633a 1875 Anom alies and curiosities of m edicine : being an encyclopedic collection of rare and extraordinary cases, and of the m ost striking G ould, G eorge M. Q S 675 G 696a 1901 instances of abnorm ality in all branches of m edicine and surgery : derived from an exhaustive research of m edical literature from its origin to the present day Anom alies and curiosities of m edicine; being an encyclopedic collection of rare and extraordinary cases, and of the m ost striking G ould, G eorge M.
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